SARS-CoV-2 and Hypertension: Evidence Supporting Invasion into the Brain Via Baroreflex Circuitry and the Role of Imbalanced Renin-Angiotensin-Aldosterone-System

This study explores the hypothesis that SARS-CoV-2 infection may contribute to hypertension by affecting the central nervous system, particularly through invasion into the brain via baroreflex circuitry, and by disrupting the balance of the renin-angiotensin-aldosterone system (RAAS). Key Findings: In summary, this study provides a comprehensive examination of how SARS-CoV-2 may contribute to hypertension through its effects on the CNS and RAAS. It highlights the complex interplay between viral infection, immune response, and cardiovascular regulation, suggesting new avenues for understanding and potentially treating hypertension in COVID-19 patients. Read More: